Dr Seyfried addresses these fuel sources with his two-fold cancer therapy - with ketosis (withholding glucose) AND a glutamine (amino acid) suppressor medication. He explains this over and over. Some people aren’t listening.
Feeding all cells not only cancer cells
this is worth listening to over and over.
Jay, can you do a video about diet for people with chronic kidney disease, including non-diabetic CKD? Not how to prevent it but what to do once you have it? There is no information on this except mainstream diet advice which I don't trust.
Right from the start, this "expert" gets it wrong: "Mitochondria converts from Oxidative phosphorylation to produce to ATP, it uses glycolysis fermentation" This is incorrect—glycolysis actually occurs in the cytosol, not the mitochondria. The cytosol is the liquid matrix surrounding the organelles. Pyruvate, the end product of glycolysis, offers cancer cells multiple metabolic options. Some pyruvate is converted to lactate and expelled from the cell, while a significant portion can be THEN transported into the mitochondria, where it undergoes further metabolism. Glycolysis is an anaerobic process, meaning it doesn’t require oxygen. While a small amount of ATP can be made in mitochondria without direct oxygen involvement, the bulk of mitochondrial ATP generation (via oxidative phosphorylation) absolutely requires oxygen.
Wait, according to Dr. Seyfried, cancer can't use Ketones for energy. What is truth here? Is taking exogenous ketones for cancer a bad idea?
Jay Feldman misses the mark here: "A high NAD+/NADH ratio indicates a good metabolism, while a low ratio suggests poor metabolism, and in every degenerative state, including cancer, we see a lower NAD+/NADH ratio." While it’s true that a high NAD+/NADH ratio can be linked to optimal mitochondrial function, the paradox is that cancer cells often show a higher NAD+/NADH ratio than normal cells. This isn’t entirely surprising, as increased NAD+ levels fuel glycolysis, which many cancer cells depend on, and can actually contribute to mitochondrial dysfunction. Elevated NAD+ also inhibits apoptosis, giving cancer cells a survival advantage.
Are yall really that ignorant
@WalkinginDivineHealth