@simonwiltshire7089
Think you are going to have to discuss this idea with Ben Bickman.
@binathere2574
Yes you are mechnics. Human body mechanics. Thank you. I love your information.
@SewDecorous
I would love to hear your take on how iron deficiency (even without anemia) can affect A1C. A year ago, my A1C jumped out of nowhere to 5.7 (was 5.4 3 months prior) but no other markers indicated pre-diabetes…good fasting glucose, triglycerides in the 50-80 range, etc. But then I read that iron deficiency even without anemia can cause falsely high A1C. Only my MCHC marker hinted at falling iron levels. It was steadily declining and went to just out of range on the low side. I started taking beef liver and organ supplements around July and finally my A1C was down to 5.6 at my blood draw in September and hopefully still dropping. I do have gluten issues and was glutened pretty badly before the rise and once during. I blame that for my iron dipping low.
@sebastianbaran9645
The Ray Peat diet caused me to develop type II diabetes, but thankfully, I reversed it by switching my carbohydrate sources to beans and vegetables and getting my fat primarily from unsaturated sources like olive oil, avocados, and nuts and seeds (not seed oils).
@kjartanthorottarsson2731
Would love to hear your discuss Dr. David Stephens glucose protocol. It's quite in line with bioenergetic ideas but different in practice to what Ray recommended.
@sharkair2839
impaired glucose metabolism i love it.
@markhapner8499
Thanks for your dive into the details of impaired glucose oxidation and the cell's fallback to lipid oxidation and glycolysis. Unfortunately, there doesn't seem to be research that has uncovered the causes of this metabolic impairment. You mention the research found that lean individuals had healthy glucose metabolism; however, it is well known that the number of TOFI (those who are thin with fatty organs) is also significantly increasing and has resulted in many thin with T2D. Whatever is causing impaired glucose metabolism is likely highly associated with the increase of visceral fat. You both may want to provide more info on this as well as info on what may be driving this overall descent into impaired metabolism. If you think you have some clues that should be investigated, you should send a note to Jay Bhattacharya who will soon be cleaning house at the NIH. Hopefully, he will be redirecting its vast healthcare research budget to areas that will address the causes and actual cures of our chronic disease epidemic. I think he knows that GLP1 agonists is not the solution :>).
@solomani-42
Would reducing fat at the same time increasing carbs help fix this problem … or not really as fat is actually needed for a healthy metabolism?
@fille.imgnry
What are some other sources of information around the bioenergetic approach? I would love to listen to some discussion with other people that are more into the ketogenic diet or simply would claim that sugar is problematic.
@firashammad5775
What would you recommend for a T2 diabetic patient with A1C of 9.0 to do about it,I eat medium fat and medium carb diet .
@JEHOVAH485
And how do you get the cells to use energy better then?
@felipearbustopotd
How much does muscle mass matter? If you have low muscle mass, would that limit the amount of glucose receptors that could be used to soak up any excess?
@jberts3141
Hey Jay im trying to find the video where you or mike talk about how mitochondria spin water to create atp and if the membrane is made out of unstable fat like PUFAs the water leaks out and cant generate efficiently?
@eplenishing
Would this apply to myalgic encephalomyelitis too?
@8_bit_Geek
Seems like the core problem is still sugar. It’s needed and long term keto or carnivore has its own issues. But this sounds like people eat too many carbs and store too much fat from too little energy expenditure and your video makes it sound like t2d is a survival mode to only burn fat because the body is close to max storage and people still keep eating carbs, etc Probably a genetic factor too with the ability to store fat. Some people can store more and don’t get t2d and others reach max fat storage and go into diabetes Would be interesting to correlate with historical ldl values. Imagine people get high ldl to try to burn more fat to keep out of t2d mode and get a prescription for statins which does the opposite and pushes the body into t2d
@jx2938
👏
@Aetherfield
What happens when a non-diabetic but impaired “pre-diabetic” receive insulin? Will that help their mitochondria function better?
@Veritythree60
🔥🔥🔥
@firelight-vitality